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CDK1 directly binds and phosphorylates the S447 site of ACSL4, promoting the ubiquitination degradation of ACSL4, while genetic and pharmacological blocking of CDK1 enhances ACSL4 activity, promoting ferroptosis of colon cancer cells and increasing their susceptibility to oxaliplatin

Khrenov Journal: Archives of biochemistry and biophysics Journal ranking: Q1 Key takeaways: Thymulin and peroxiredoxin 6 improve blood-brain barrier conditions and restore symptoms in mice with experimental multiple sclerosis, offering potential treatment strategies for multiple sclerosis

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If they were persistent or severe, the parents were offered the option to discontinue the study