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MEDVi strengths align well with certain patient profiles while being less ideal for others

Together, they create a more powerful metabolic effect than either receptor alone, which explains why tirzepatide outperforms semaglutide in clinical comparisons

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Pro-inflammatory cytokines increase neuronal vulnerability to apoptosis and oxidative stress by activating enzymes such as JNK, ERK, dsRNA-dependent protein kinase (PKR), and IB kinase (IKK) that reduce insulin sensitivity by serine phosphorylation of IRS1 [144, 145]