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In Zucker diabetic fatty (ZDF) rats, a model characterized by spontaneous obesity, hepatic steatosis, and insulin resistance, EA significantly reduced fasting plasma glucose, insulin, C-peptide levels, and the homeostatic model assessment for insulin resistance (HOMA-IR) index (47)

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Keap1-null mutation leads to postnatal lethality due to constitutive Nrf2 activation

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