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Using in vivo, ex vivo, and in vitro models, we now show that the mechanism of sustained hyperinsulinemia in the setting of FATP2 inhibition or deletion was cellmediated GLP-1 secretion with paracrine stimulation of cell insulin secretion

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Collected CMs were used to assess their ability to induce capillary-like structures of HUVE cells on matrigel

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Over the next few years he and his collaborators at NIH identified several new peptides and hormones they published in obscure journals

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