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Naturally, this remains a plausible pathophysiological hypothesis, not a causal demonstration derived from the study

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Research has focused on its facilitation of synapse formation via HGF/Met receptor signalling, with investigations suggesting neurotrophic potency orders of magnitude greater than BDNF in dendritic branching assays

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A2A adenosine receptor overexpression and functionality, as well as TNF- levels, correlate with motor symptoms in Parkinsons disease

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10.1073/pnas.1322003111 177 YanceyP

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