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Despite the existing body of reviews describing ferroptosis in the context of diabetes pathophysiology and complications ( FIGURE 1 2 The mechanism of ferroptosis in diabetes-associated cognitive dysfunction Iron, an essential trace element, crosses the blood-brain barrier via a complex metabolic regulatory network and participates in diverse critical biological processes within the central nervous system, including oxygen transport, energy metabolism, and the synthesis and catabolism of myelin and neurotransmitters (Table 1)

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Recently, various high-throughput-based omics studies have been used to screen for NP toxicity, including genomics, transcriptomics, proteomics, metabolomics, etc., (Matysiak et al., 2016

doi: 10.1038/srep09102 30 WangSWeiJFanYDingHTianHZhouXet al

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