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The proposed mechanism centers on modulation of the nitric oxide system: BPC-157 counteracts L-NAME-induced eNOS blockade in rat models and supports vascular sprouting through VEGFR2 internalization (Hsieh et al., J Mol Med , 2017)

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The role of S-glutathionylation in health and disease: a birds eye view

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(2019) "MOTS-c nuclear translocation under metabolic stress." Published in Cell Metabolism