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Combined intestinal loss of PPAR// attenuates KD-driven intestinal stem cell expansion, proliferation and clonogenicity, whereas inhibition of downstream fatty acid oxidation through CPT1A loss limits adenoma formation specifically under KD, linking tumour initiation to fatty acid oxidation of dietary lipids rather than lipid accumulation

Cell Death Discov 10:45

The fluid is then examined under a microscope for urate crystals

In particular, 4IgB7-H3 is restricted to glioblastoma cells and may serve as a target for therapy, while 2IgB7-H3 presents with higher expression in recurrent glioblastoma and increases resistance to temozolomide (122)

TN, AJS and SEW performed statistical analyses

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