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Energy Expenditure and Thermogenesis GLP3s glucagon receptor activation specifically contributes to increased energy expenditure through multiple pathways[6]: Enhanced hepatic fatty acid oxidation and lipolysis Increased thermogenic activity in brown adipose tissue Upregulation of uncoupling protein expression Potential FGF21-mediated metabolic effects This thermogenic component distinguishes the blend from pure GLP-1 or amylin-based approaches, addressing both energy intake (via satiety) and energy expenditure simultaneously

This finding is in agreement with clinical findings that human nucleus pulposus (NP) fibrosis is essential for the maintenance of IVD height and mechanical properties in patients following percutaneous endoscopic lumbar discectomy (PELD)

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