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PMID 17619527
These medications differ in their mechanisms, administration routes, and clinical applications
Compared with untreated individuals, those receiving GLP-1 therapy showed lower levels of pro-inflammatory cytokines (IL-6, TNF-, IL-12), higher levels of IL-10, reduced expression of endothelial adhesion molecules (ICAM-1, VCAM-1), and decreased leukocyte rolling and adhesion along the vascular endothelium [15]
In addition to altering food satiety signals, GLP-1 is suggested to modulate satiety in drugs of abuse and alter reward-related changes in multiple drugs of abuse such as cocaine (Hernandez et al., Even though the role of GLP-1 on neuroprotection is well-known and new clinical trials have been started for its repurposing in neuroprotection, the role of GLP-1 on reward systems and its potential clinical use for reward modulation still need further studies
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