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Within this metabolic context, GLP-1RA therapy may create conditions in which reliance on lipid-derived substrates increases the functional importance of redox-buffering systems that maintain oxidative stability
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2.2.2 ERK1/2-HDAC6 signaling pathway The GLP-1R-ERK1/2-HDAC6 signaling pathway complements the mitophagy pathway by modulating oxidative stress and autophagy
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Patients who maintained muscle mass experience a much smoother metabolic transition after stopping
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