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While GHSR1a constitutive activity is required for amplification of MC3R signaling, it has minimal effect on GHSR1a-MC3R dimer internalization, as treatment with the GHSR1a inverse agonist SP-analog only results in a small, nonsignificant reduction in internalization (Schellekens et al., 2013)

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Differences were analyzed using Student's t-test or one-way analysis of variance, followed by Tukey's post hoc test

CJC-1295 WITH DAC = long-acting (lasts 68 days), inject 12x/week

Reported characteristics of the individual components are described strictly as observations made in published in vitro and animal-research contexts

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