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Sterile inflammatory response in acute pancreatitis

J.PuderM

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If it was really and truly a hormone that could increase glucose-dependent insulin secretion, then it had potential to treat diabetes

Instead, it acts upstream at the level of neural desire circuits

This apparent contradiction may reflect a critical evolutionary adaptation in which cancer cells reprogram lactate metabolism to attain dual benefits (enhanced metabolic flexibility coupled with augmented oxidative stress resistance) thereby facilitating survival under adverse microenvironmental conditions