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This orexigenic drive appears to be largely GH-independent, as it is observed even in models of GH-deficiency, and is accompanied by rises in leptin that likely reflect expansion of adipose depots and provide a feedback brake on further hyperphagia

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found that ferroptosis in ectopic endometrial stromal cells in patients with ovarian endometriosis could activate the p38 mitogen-activated protein kinase (p38 MAPK)/signal transducer and activator of transcription (STAT) 6 signalling pathway, thereby promoting local upregulation of vascular endothelial growth factor A (VEGFA) and interleukin-8 (IL-8) in ectopic lesions [15]

PMID: 19 SeoG-YTakahashiDWangQMikulskiZChenAChouT-Fet al