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Cellular senescence in ovarian somatic cells, including GCs and stromal cells, may further exacerbate oxidative stress and inflammatory signaling within the follicular niche [80, 81, 82] Together, these interconnected mechanisms and hallmarks of aging suggest that reproductive aging shares common biological pathways with systemic aging, supporting the view that oocyte aging should be considered within a broader geroscience context

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Understanding glutamine depletion across various etiologies is crucial, especially in patients with AKI stage 3 undergoing RRT

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