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Evidence from preclinical and clinical studies highlights their potential in reducing psoriasis area and severity index (PASI) scores, mitigating immune hyperactivity, and enhancing the safety and efficacy of standard therapies

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A prime example is the DHHC12-catalyzed palmitoylation of NLRP3, which increases its affinity for the chaperone heat shock cognate 71 kDa protein (HSC70) and promotes its degradation via chaperone-mediated autophagy, thereby preventing excessive inflammation [38, 143]