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Such alterations were also associated with inhibition of cytochrome c oxidase (CcOX) activity and microtubule network disassembly, which are considered hallmarks of nitric oxide (NO) toxicity, indicating that NO, rather than the depletion of GSH per se, is the primary mediator of cell damage

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This can lead to an accumulation of those reactive intermediates, which may cause stress to liver cells over time

But vitamin E, molybdenum and possibly other metals can weaken absorption