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In particular, mitochondrial uncoupling has emerged as a promising strategy due to its ability to disrupt metabolic flexibility, induce energetic stress, and alter cellular redox regulation in cancer cells

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Although cancer cells typically show a metabolic shift toward increased glycolysis and reduced oxidative phosphorylation, a metabolic adaptation known as the Warburg effect, they still depend on mitochondrial ATP production for survival (Weinberg and Chandel, 2015

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