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Prolonged use of EGFR/ALK tyrosine kinase inhibitors in non-small cell lung cancer, for instance, can reprogram the local immune milieu, polarizing macrophages toward an immunosuppressive M2 phenotype and upregulating barrier proteins, which collectively impair T cell function [116]

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11 Structural studies of cagrilintide bound to these receptors show that its lipid tail and stabilized helix create distinctive, long-residence interactions at the receptor interface

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Dans SUSTAIN-6, aucune de ces ractions na t considre comme svre, et aucun cas na conduit larrt du traitement