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The accumulation of AGEs by itself also contributes to neuronal damage through the conglomeration of adipocytokines, initiating apoptosis and accelerating nerve fibre degeneration

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and 4) Oxidative stress and NO bioavailability, since age-related oxidative stress and downregulation of endothelial nitric oxide synthase (eNOS) impair the bioavailability of NO[293], which is likely to contribute to decreased microvascular density

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