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Nevertheless, we found that a small fraction (~10%) of condensed, ciprofloxacin-treated cells briefly became turgid, as indicated in phase-contrast by phase-light regions becoming phase-dark and increases in cell size, so that hypoosmotic shock appeared to transiently reverse cytoplasmic condensation in these cells (Fig

Role of epidermal growth factor receptor signaling in the interaction of Neisseria meningitidis with endothelial cells