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Moreover, stimulation of GLP-1R in BMSCs results in the accumulation of -catenin in the cell nucleus

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The CVOTs provide additional evidence for cardiovascular risk reduction with GLP-1RAs in patients with T2D

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It is tempting to speculate that this apparent loss of benefit in patients with more advanced HF may be the result of increases in HR caused by activation of sinoatrial node (SAN) GLP-1Rs (64, 65), which could be mitigated with an agent such as GLP-1(2836) that retains cardiac and vascular protection of GLP-1, without activating the GLP-1R

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