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But with age, these may become dysfunctional, which highly impairs muscle recovery after an injury

However, the exact antiarrhythmic mechanism of flecainide remains a topic of debate and may involve a combination of sodium channel block (preventing triggered activity), inhibition of RyR2 (reducing the likelihood of spontaneous SR calcium-release events), or indirect effects on intracellular calcium handling due to changes in intracellular sodium [67, 68]

It is an essential cofactor for the enzymes involved in the synthesis of vital hormones and monoamine neurotransmitters, in particular catecholamines (dopamine, adrenaline, noradrenaline) and indolamines (serotonin and melatonin)