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It has attracted significant scientific interest due to its apparent stability in harsh gastric environments and its role in tissue protection and repair-related research pathways

Acetaminophen and vigabatrin deplete glutathione stores leading to loss of feedback inhibition and increased production of 5-oxoproline from its precursor gamma glutamylcysteine

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However, at Johns Hopkins University, researchers met these standards by evaluating members of the Boston Birth Cohort [3]

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