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The phosphatidylinositol 3-kinase (PI3K)/protein kinase B (AKT)/mTOR pathway can be activated by PIK3CA mutation, loss of phosphatase and tensin homolog (PTEN), or other mechanisms (e.g., PIK3CB mutation, PIK3CB mutation, AKT1 mutation, TSC1/TSC2 inactivating mutation, MTOR mutation) to promote PCa cell survival and proliferation independent of AR signaling (33)

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Jun 2004;10(6):Ra125-31