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Fourth, a number of patients withdrew from the study but no causal association can be determined

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(2012) 2:e134

These findings highlight the critical role of THC-induced oxidative stress as a contributing factor to cannabinoid-mediated neuropsychiatric risk and identifies a novel antioxidant treatment candidate for the prevention and/or reversal of these pathophysiological outcomes

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lotspecific analytical data are provided on the certificate of analysis (COA)