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Discussion Elevated levels of plasma lactate, ketones and uremia are common causes of HAGMA

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As chronic inflammation and mitochondrial dysfunction can result in oxidative stress (54, 55, 56, 57) and our results show that human inflammatory PE placentas exhibit more oxidative DNA damage (Fig 1I and J), we next investigated whether LPS treatment leads to placental oxidative DNA damage in this model

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In contrast, the positioning of mitochondria (stained for cytochrome oxidase IV (Cox IV)), EEA1-positive early endosomes, and RAB8-positive endosomes was unaltered by the KO of either TMEM55B or TBC1D9B (Supplementary Fig