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Tab 3: TCR-T monoculture surfaceomes

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The Proposed GLP-1 Agonist POP Toxin Redistribution Axis In Short Bioaccumulation of POP chemicals into adipose fat from normal to high exposures GLP-1 agonist drug induces rapid adipose fat burning Rapid lipolysis of adipose fat releases stored POPs into the bloodstream in high amounts, and over prolonged periods of time POPs recirculate bound to lipoproteins and to a lesser extent, albumin With the substantial loss of adipose fat, POP toxins are not adequately bio-transformed, but instead redistributed to other lipid-rich organs, cells and tissues Abstract Research studies on lipophilic, persistent organic pollutants (POPs) such as Dioxin, PCB, PBDEs and HCB have demonstrated that these types of toxins are predominantly stored in adipose fat

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