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However, recurrent bouts of such inflammation and the toxic milieu that ensues, are thought to produce cumulative damage to myelin, oligodendrocytes and neurons, leading to progressive degeneration, atrophy and increasing disability, exacerbated by failure of myelin repair

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These processes enable vitamin D to regulate calcium and phosphorus homeostasis and exert genomic and non-genomic actions that impact a wide variety of physiological functions [27]

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Genetic control of programmed cell death in Drosophila melanogaster