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Kidney aging in diabetic individuals is the cumulative effect of cellular senescence, advanced glycation end product accumulation, RAAS dysfunction, inflammation, and oxidative stress

In fact 30% of the oxidation of glucose in the liver occurs via the PPP

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Trpm2 deficiency in microglia attenuates neuroinflammation during epileptogenesis by upregulating autophagy via the AMPK/mTOR pathway