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Critically, data must be interpreted in a compartment-aware manner and paired with source attribution (e.g., NOX versus mitochondrial origin) using targeted reporters, pharmacologic/genetic controls and site-specific sampling to avoid conflating adaptive signalling with downstream injury (Murphy et al

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Antibodies against DCN or AGER shielded Gpx4 knockout mice from cerulein-triggered pancreatitis, suggesting an indirect influence of ferroptosis-released DAMPs on immune cells [102]