On the one hand, its activation can promote cancer progression, invasion, metastasis, and resistance to chemotherapeutic agents [68]
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Research involving GHK-Cu commonly investigates its interaction with: Collagen- and elastin-related pathways Glycosaminoglycan (GAG) and extracellular-matrix signaling Tissue-maintenance and dermal-support mechanisms Angiogenesis-related pathways Vascular-support and circulation-related signaling Antioxidant-defense systems, including superoxide dismutase (SOD)-related activity Research commonly explores how copper-related signaling pathways may interact with cellular-renewal, tissue-remodeling, and physiological-maintenance mechanisms
doi: 10.1016/j.cell.2017.03.035 105 GuYAlbuquerqueCPBraasDZhangWVillaGRBiJet al
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