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These metabolic abnormalities further influence immune cell metabolism and neuronal function through signaling byproducts such as lactate and ROS, thereby potentially linking metabolism, immunity, and neurobiology in the pathogenesis of PEM (102)

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Elevated levels of TNF and IL-1, in part through the release of prostaglandin E2, contribute to excitotoxicity and synaptic loss [28, 29]

excessive use may cause imbalances or mild adverse reactions

What the Research Is Telling Us Two studies from 2024 have raised big red flags: A study published in JAMA Ophthalmology found that semaglutide users were over four times more likely to develop NAION than those on other diabetes drugs