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The real differentiator may be the metabolic preservation effects seen with CagriSema in preclinical models, but human data confirming this advantage is still limited

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Abdominal obesity-induced insulin resistance and hyperinsulinemia exacerbate uric acid reabsorption, facilitated by the sodium and monocarboxylate transporter (SMCT) exchanging with URAT1, leading to an upsurge in serum uric acid levels