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Targeting key inflammatory cytokines and pathways, such as IL-1, TNF-, and NF-B, is a primary strategy to mitigate liver damage and systemic inflammatory response syndrome [29-32]

Further studies showed that IGF-1 specifically induced NF-B activity

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Increasing evidence indicates that these abnormalities are not independent phenomena but components of a self-perpetuating redox-endocrine network that sustains hyperandrogenism, anovulation, and metabolic impairment

Condicin: Fine

Topical application is considered the least problematic solution if used correctly